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glutathione reductase gssg Mitochondrial in Cellular Redox Homeostasis and Disease Manifestation Volume:150 ml

Kampf gegen Krebs: Luxemburger Forscher präsentieren bahnbrechende Studie

Hou et al., 2016)

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Volume:150 ml

As indicated above, the reason for the loss of lysosomal hydrolytic function may not be a result of deficiency in LAL expression as atherosclerosis progresses but rather a decrease in the catalytic activity of LAL as a result of excess lysosomal cholesterol accumulation from modified forms of LDL (Figure 3)

glutathione reductase gssg Mitochondrial in Cellular Redox Homeostasis and Disease Manifestation Volume:150 ml

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